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The first published report of a female with a 45,X karyotype was in by Dr. The most commonly observed are congenital obstructive lesions of the left side of the heart, leading to reduced flow on this side of the heart. Calcification also occurs in the valves, [25] which may lead to a progressive valvular dysfunction as evidenced by aortic stenosis or regurgitation. Services include an evaluation to identify genetic diseases, chromosomal analysis, amniocentesis for prenatal diagnosis, molecular and DNA diagnostics, genetic counseling. Heart defectsdiabeteslow thyroid hormone [1]. Most Turner syndrome patients are employed as adults and lead productive lives. Women with Turner syndrome are almost universally infertile.

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Turner syndrome occurs in between one in 2, [6] and one in 5, females at birth.

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Normal skeletal development is inhibited due to a large variety of factors, mostly hormonal. Turner syndrome may be diagnosed by amniocentesis or chorionic villus sampling during pregnancy. Most Turner syndrome patients are employed as adults and lead productive lives. There is no way to remove the extra X chromosome. Approximately 99 percent of fetuses with Turner syndrome spontaneously terminate during the first trimester.

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